Foot and ankle covers the forefoot — hallux valgus, hallux rigidus, lesser toe deformities, metatarsalgia and Morton’s neuroma — the diabetic foot and Charcot neuroarthropathy, arthritis of the ankle and hindfoot, tendon disorders and the adult acquired flatfoot, and the cavus foot, ankle instability and sports injuries. Much of it rests on understanding deformity: which joints are flexible and which are fixed, what is driving the deformity, and therefore whether a soft-tissue procedure, an osteotomy or a fusion is appropriate.
100 questions in the bank · 2 topics Updated 11 November 2026
Hallux valgus questions give the radiographic angles and the state of the first metatarsophalangeal and tarsometatarsal joints, and ask which type of procedure fits.
Flatfoot questions are staged: is the hindfoot deformity flexible or rigid, can the patient perform a single-leg heel raise, and is the ankle involved?
Cavus foot questions test the Coleman block test and the search for a neurological cause.
Diabetic foot questions ask you to separate Charcot neuroarthropathy from infection and to know the principles of offloading.
Ankle arthritis questions ask for the factors that favour fusion or replacement, and the position in which an ankle is fused.
Classic traps and discriminators
The Coleman block test shows whether hindfoot varus is driven by a plantarflexed first ray: if the heel corrects to valgus with the first ray hanging off the block, the hindfoot is flexible and the deformity is forefoot-driven.
A unilateral or rapidly progressive cavus foot is not assumed to be idiopathic: it needs a neurological examination and imaging of the spine.
In an acutely hot, swollen, neuropathic foot with intact skin, Charcot neuroarthropathy is more likely than osteomyelitis; diabetic foot infection almost always begins from an ulcer.
An arthritic first metatarsophalangeal joint with hallux valgus is treated by fusion, not by a realignment osteotomy.
An Achilles tendon rupture is diagnosed clinically — a palpable gap and absent plantarflexion on calf squeeze — and active plantarflexion can be preserved by the other flexors, so its presence does not exclude rupture.
Free foot & ankle practice questions
From the free sample — original, referenced, with the full explanation. Rate your confidence first.
A weight-bearing radiograph of a 50-year-old woman with hallux valgus shows the sesamoids lying lateral to the first metatarsal head. In fact the sesamoids have kept their relationship to the second metatarsal while the metatarsal head has drifted medially off them.
Which structures tether the sesamoid complex in this position?
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Explanation
In hallux valgus the sesamoids do not sublux: the metatarsal head moves medially off the sesamoid apparatus once the medial capsule and medial sesamoid ligament attenuate and the crista erodes. The fibular sesamoid stays anchored to the second ray by the adductor hallucis and the deep transverse metatarsal ligament, which is why these are divided in a lateral release. Abductor hallucis migrates plantarwards and loses its abducting action, allowing pronation, but it is a medial structure. Flexor hallucis longus bowstrings laterally with the sesamoids as a deforming force and is not the tether. The medial sesamoid ligament is the structure that fails. Extensor hallucis brevis is dorsal. Reference: Coughlin and Mann's Surgery of the Foot and Ankle 9e; Miller's Review of Orthopaedics 8e.
A 58-year-old man is to have an ankle arthrodesis for post-traumatic arthritis. The hindfoot and midfoot joints are mobile and free of arthritis.
In which position should the talus be fused to the tibia?
How sure are you?
Explanation
The accepted position for ankle arthrodesis is neutral in the sagittal plane, about 5° of hindfoot valgus and 5–10° of external rotation matching the other side, with the talus translated slightly posteriorly under the tibia to shorten the lever arm of the foot and ease roll-over. Equinus produces a vaulting gait, a recurvatum thrust at the knee and midfoot overload. Varus locks the transverse tarsal joint, giving a rigid foot that bears weight on its lateral border. Internal rotation is poorly tolerated because the foot trips the swing leg, and excessive external rotation strains the knee and medial foot. Anterior translation lengthens the lever arm. Fixed dorsiflexion overloads the heel. Reference: Buck P, Morrey BF, Chao EY, J Bone Joint Surg Am 1987; Miller's Review of Orthopaedics 8e.
What are the Eichenholtz stages of Charcot neuroarthropathy?+
Stage I is development or fragmentation: a hot, swollen, red foot with fragmentation and subluxation on radiographs. Stage II is coalescence: the swelling and warmth settle and the fragments begin to consolidate. Stage III is reconstruction or consolidation: a cool, stable foot with residual deformity. A prodromal stage 0, with clinical signs and normal radiographs, was added later.
How is adult acquired flatfoot staged?+
Classically by the state of the tibialis posterior tendon and the flexibility of the deformity: stage I is tenosynovitis without deformity; stage II is a flexible planovalgus foot in which the patient cannot perform a single-leg heel raise; stage III is a rigid hindfoot deformity; stage IV adds valgus tilt of the talus from failure of the deltoid ligament. Flexible deformities are treated by tendon transfer and osteotomy, rigid ones by fusion.
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